Implantable cardioverter-defibrillator (ICD) use is associated with reduced short- and long-term mortality in patients with heart failure, according to late breaking research presented at ESC Congress 2019 together with the World Congress of Cardiology1 and published in Circulation.
Beta-blockers remain effective for preventing death in heart failure with reduced ejection fraction (HFrEF) and sinus rhythm, even in patients with moderate or moderately-severe kidney dysfunction, according to late breaking research presented in a Hot Line Session today at ESC Congress 2019 together with the World Congress of Cardiology.
Elderly patients with heart failure who see a pharmacist once a week are more likely to take their tablets and be active in daily life, according to late breaking results from the PHARM-CHF randomised controlled trial presented today at Heart Failure 2019, a scientific congress of the European Society of Cardiology (ESC).1
Heart failure patients who consume more dietary fibre tend to have healthier gut bacteria, which is associated with reduced risk of death or need of a heart transplant. The fibre study was presented today at Heart Failure 2019, a scientific congress of the European Society of Cardiology (ESC).1
Heart Failure with Preserved Ejection Fraction (HFpEF) especially affects women and the elderly. The authors review the importance of the heterogeneity of this condition and its implications on therapeutic approaches. Although there are no sex-specific recommendations regarding the evaluation and management of HFpEF in women compared with men, women are more likely to have the HFpEF phenotype than HFrEF.
Researchers at the University of Arizona have discovered that metformin, a drug commonly used to treat type 2 diabetes, might also be used to treat heart failure with preserved ejection fraction (HFpEF), a condition that is predicted to affect over 8% of people ages 65 or older by the year 2020. The study, which was published in the Journal of General Physiology, shows that metformin relaxes a key heart muscle protein called titin, allowing the heart to properly fill with blood before pumping it around the body.